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Conclusion: SYNE1 genetic variants are associated with a spectrum of neurological disorders, particularly SYNE1 ataxia, which presents with both cerebellar and noncerebellar symptoms

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Notably, schlank, the drosophila CERS isoform, and mammalian CERS2 are capable of responding to intracellular fatty acid levels with direct binding and upregulation of Srebf expression and, conversely, downregulated expression of intracellular lipases (74, 75)